TY  - CHAP
M1  - Book, Section
TI  - Pneumoconiosis
A1  - Khattar, Ramni
A1  - Vlahovich, Kevin
A1  - Sood, Akshay
A2  - Boulton, Matthew L.
A2  - Wallace, Robert B.
PY  - 2022
T2  - Maxcy-Rosenau-Last Public Health & Preventive Medicine, 16e
AB  - Pneumoconiosis  is  a  general  term  for  a  collection  of  lung  diseases  caused  by  the  inhalation  and  accumulation  of  usually  inorganic  dust  particulates,  generally  from  the  workplace,  and  eliciting  an  injurious  reaction  of  the  lung  tissue.  Inhalational  dust  exposure  can  lead  to  benign  and  malignant  diseases  of  the  lung  parenchyma,  airway,  and  pleura.  In  this  chapter,  we  outline  the  nonmalignant  respiratory  effects  related  to  inhalational  exposure  to  coal  mine  dust,  asbestos,  and  silica;  metals  such  as  uranium  and  cobalt;  as  well  as  man-made  vitreous  fibers.  As  shown  in  Fig.  E1-1,  parenchymal  pneumoconiosis  may  be  classified  as  nodular  lung  diseases  (including  progressive  massive  fibrosis  or  PMF),  dust-related  diffuse  pulmonary  fibrosis  (or  secondary  usual  interstitial  pneumonitis  or  UIP),  other  interstitial  pneumonitis  (including  hypersensitivity  pneumonitis  from  organic  components  of  the  dust),  and  alveolar  proteinosis.  Airway-dominant  pneumoconiosis  may  be  classified  as  the  chronic  bronchitis  and/or  emphysema  phenotypes  of  chronic  obstructive  pulmonary  disease  (COPD),  work-related  asthma,  and  small  airways  diseases.  Pleural  changes  of  pneumoconiosis  include  pleural  effusions,  rounded  atelectasis,  circumscribed  pleural  thickening/pleural  plaques,  and  diffuse  pleural  thickening.  This  chapter  not  only  focuses  on  the  nodular  and  fibrotic  parenchymal  lung  diseases,  but  also  considers  the  other  protean  manifestations  of  pneumoconiosis.  
SN  - 
PB  - McGraw Hill
CY  - New York, NY
Y2  - 2024/04/19
UR  - accessmedicine.mhmedical.com/content.aspx?aid=1188599337
ER  -