TY  - CHAP
M1  - Book, Section
TI  - Occupational Asthma, Byssinosis, and Industrial Bronchitis
A1  - Cooper, J. Allen D.
A2  - Grippi, Michael A.
A2  - Elias, Jack A.
A2  - Fishman, Jay A.
A2  - Kotloff, Robert M.
A2  - Pack, Allan I.
A2  - Senior, Robert M.
A2  - Siegel, Mark D.
PY  - 2015
T2  - Fishman's Pulmonary Diseases and Disorders, 5e
AB  - Inhalation  of  dusts,  fumes,  and  organic  substances  at  the  workplace  can  cause  a  number  of  pulmonary  syndromes.1–4  The  lung  parenchyma  and  airways,  as  well  as  the  pleura,  can  be  affected  by  inhalation  of  foreign  substances.  This  chapter  discusses  reactions  of  the  large  airways  to  inhalation  of  toxic  substances  present  in  the  workplace.  Lung  parenchymal  and  pleural  reactions,  as  well  as  obliterative  bronchiolitis  in  response  to  inhaled  materials,  are  discussed  elsewhere  in  this  text.  Occupational  airway  disease  can  manifest  itself  as  chronic  bronchitis  with  variable  airway  hyperreactivity  (industrial  bronchitis  or  asthma-like  syndrome),  or  with  asthma  accompanied  by  persistent  hyperreactivity  of  the  airways  (occupational  asthma).  Some  occupational  exposures  can  cause  both  industrial  bronchitis  and  asthma  whereas  others  cause  only  one  or  the  other.  Cotton  dust  is  the  most  common  cause  of  industrial  bronchitis  without  occupational  asthma.  Grain  dust  can  cause  both  industrial  bronchitis  and  asthma.  In  this  chapter,  general  and  specific  issues  regarding  industrial  bronchitis  and  occupational  asthma  are  discussed.  
SN  - 
PB  - McGraw-Hill Education
CY  - New York, NY
Y2  - 2024/04/18
UR  - accessmedicine.mhmedical.com/content.aspx?aid=1122364707
ER  -