The patient's clinical features are consistent with septic shock. In hypovolemic or cardiogenic shock, patients will have increased heart rate and decreased blood pressure, but will have cold skin. Septic shock causes a generalized vasodilation, producing the symptoms. However, in response to hypovolemic or cardiogenic shock, the peripheral arteries will constrict to facilitate shunting of blood to the vital organs and cause cold extremities. Acute respiratory distress syndrome (ARDS) is a common complication of septic shock, and is characterized by development of bilateral infiltrates and consolidation on chest radiograph. There is no evidence that this patient has suffered a myocardial infarction (A). Central venous pressures would be expected to be low, secondary to vasodilation in the setting of septic shock. The stress response, the infection, and this patient's diabetes mellitus all make hyperglycemia more likely than hypoglycemia.