TY  - CHAP
M1  - Book, Section
TI  - Approach to Supraventricular Tachyarrhythmias
A1  - Michaud, Gregory F.
A1  - Stevenson, William G.
A2  - Jameson, J. Larry
A2  - Fauci, Anthony S.
A2  - Kasper, Dennis L.
A2  - Hauser, Stephen L.
A2  - Longo, Dan L.
A2  - Loscalzo, Joseph
PY  - 2018
T2  - Harrison's Principles of Internal Medicine, 20e
AB  - Supraventricular  tachyarrhythmias  originate  from  or  are  dependent  on  conduction  through  the  atrium  or  atrioventricular  (AV)  node  to  the  ventricles.  Most  produce  narrow  QRS-complex  tachycardia  (QRS  duration  <120  ms)  characteristic  of  ventricular  activation  over  the  Purkinje  system.  Conduction  block  in  the  left  or  right  bundle  branch  or  activation  of  the  ventricles  from  an  accessory  pathway  produces  a  wide  QRS  complex  during  supraventricular  tachycardia  that  must  be  distinguished  from  ventricular  tachycardia  (Chap.  249).  Mechanisms  of  supraventricular  tachyarrhythmia  can  be  divided  into  physiologic  sinus  tachycardia  and  pathologic  tachycardia  (Table  241-1).  Pathologic  tachycardia  can  be  further  sub-classified  in  terms  of  mechanism  as  reentrant  arrhythmias  dependent  on  AV  nodal  conduction  (e.g.,  AV  reentry),  large  reentry  circuits  within  the  atrial  tissue  alone  (e.g.,  atrial  flutter)  or  focal  atrial  tachycardias  that  can  be  due  to  automaticity  or  small  reentry  circuits  (see  Figs.  243-3  and  245-1).  The  prognosis  and  treatment  vary  considerably  depending  on  the  mechanism  and  underlying  heart  disease.  
SN  - 
PB  - McGraw-Hill Education
CY  - New York, NY
Y2  - 2024/04/19
UR  - accessmedicine.mhmedical.com/content.aspx?aid=1176803914
ER  -