- Nonsustained: Three or more consecutive QRS complexes of uniform configuration of ventricular origin at a rate of more than 100 bpm.
- Sustained: Lasts more than 30 seconds; requires intervention for termination.
- Monomorphic ventricular tachycardia.
- Polymorphic ventricular tachycardia: Beat-to-beat variation in QRS configuration.
The magnitude of ventricular tachycardia (VT), one of the most common health problems encountered in clinical practice, can best be appreciated in terms of its various clinical manifestations, which include ventricular fibrillation (sudden cardiac death [SCD]), syncope or near syncope, and wide QRS tachycardia.
The most serious is its degeneration into ventricular fibrillation, producing cardiac arrest and SCD that accounts for 200,000 deaths a year. The second most serious clinical presentation is syncope. Although the overall prevalence of VT-related syncope is unclear, it is estimated to be frequent because inducible VT (via electrical stimulation) is the most common arrhythmia detected in patients with unexplained syncope. A high prevalence of SCD (more than 20% incidence within the ensuing 12 months) is noted in patients with syncope from cardiovascular causes, suggesting that undiagnosed VT may be an underlying cause of sudden death in patients with unexplained syncope. The third most significant clinical manifestation of VT
is a wide QRS complex tachycardia that is often hemodynamically well tolerated.
Ventricular tachycardia as a cause of morbidity and mortality is grossly underdiagnosed, potentially leading to mismanagement.
This may be particularly true when the clinical presentation is
unexplained syncope because no concomitant electrocardiographic
(ECG) documentation is available. In the case of cardiac arrest
or SCD acute myocardial infarction rather than an arrhythmic problem
is often assumed to be responsible. Most persons who have suffered
sudden death have no evidence of acute myocardial necrosis, even
though the episode often occurs in patients with underlying coronary
artery disease. Managing the underlying coronary artery disease
with no regard to treating the concomitant VT is inadequate.
When hemodynamically stable VT is recorded on the surface ECG, it is often misdiagnosed as supraventricular tachycardia (SVT) with
aberrant conduction. Any subsequent management is therefore directed
toward SVT. Although the exact logic for this line of thinking is
unclear, the main reason may be that the hemodynamic stability is associated
with the broad QRS rhythm and thus the erroneous belief that the
problem cannot be VT.
The clinical presentation of VT depends on many factors, including rate, ventricular function, presence of concomitant coronary artery
disease, the presence or absence of cardioactive drugs, and even
the patient’s posture at the time of onset. Hemodynamic
tolerance of VT can, therefore, vary considerably in different situations;
at times, it can vary in the same patient, and it is prudent not
to exclude the diagnosis of VT on the basis of hemodynamic tolerance
alone. It must be understood that approximately 80% of
the patients with sustained wide QRS tachycardia have VT. To avoid
misdiagnosis, the clinician can ...