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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 37–45. Life cycle of Gnathostoma spinigerum. In the natural definitive host (pigs, cats, dogs, wild animals), the adult worms reside in a tumor that they induce in the gastric wall. They deposit eggs that are unembryonated when passed in the feces . Eggs become embryonated in water, and eggs release first-stage larvae . If ingested by a small crustacean (Cyclops, first intermediate host), the first-stage larvae develop into second-stage larvae . Following ingestion of the Cyclops by a fish, frog, or snake (second intermediate host), the second-stage larvae migrate into the flesh and develop into third-stage larvae . When the second intermediate host is ingested by a definitive host, the third-stage larvae develop into adult parasites in the stomach wall . Alternatively, the second intermediate host may be ingested by the paratenic host (animals such as birds, snakes, and frogs) in which the third-stage larvae do not develop further but remain infective to the next predator . Humans become infected by eating undercooked fish or poultry containing third-stage larvae, or reportedly by drinking water containing infective second-stage larvae in Cyclops . (Adapted from a drawing provided by Dr. Sylvia Paz Díaz Camacho, Universidade Autónoma de Sinaloa, Mexico. Content source: Global Health, Division of Parasitic Diseases and Malaria, CDC.) A flowchart of the life cycle of the Gnathostoma species.

Current Medical Diagnosis & Treatment 2024 > Gnathostomiasis

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eFigure 18–10. Schematic diagram of a percutaneous transhepatic cholangiogram, here used to provide drainage for an obstructing pancreatic carcinoma. A: A dilated peripheral intrahepatic bile duct is entered percutaneously with a fine needle (arrow). B: A long guidewire is passed through the needle into the intrahepatic duct, common hepatic duct, and common bile duct, and then advanced past the tumor into the duodenum. C: The needle is withdrawn, and a plastic catheter has been passed over the guidewire to create a channel through the liver parenchyma. D. An external-internal drainage catheter is inserted over the guidewire and through the channel, then advanced via the bile ducts and through the tumor to empty into the duodenum. (Reproduced, with permission, from James TW, Baron TH. Endoscopic and Radiologic Treatment of Biliary Disease. In: Feldman M, Friedman LS, Brandt LJ [editors]. Sleisenger and Fordtran's Gastrointestinal and Liver Disease, 2 vol set, 11th edition. Philadelphia, Elsevier; 2023.) Four diagrams of the liver and gall bladder depict the steps in a percutaneous transhepatic cholangiogram.

Current Medical Diagnosis & Treatment 2024 > Jaundice & Evaluation of Abnormal Liver Biochemical Tests

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eFigure 18–43. Hemangioma of the liver. Early post-contrast (M-7a) and late post-contrast (M-7b). Hemangiomas are the most common primary tumors of the liver, demonstrate progressive, centripetal contrast enhancement (arrows), and are uncommonly found in patients with cirrhosis. They are predominantly found in women and usually benign. (Used, with permission, from Nicholas Fidelman, MD). An axial early post-contrast image shows a tumor in a liver lobe. An axial late post-contrast image shows an opaque tumor in a liver lobe.

Current Medical Diagnosis & Treatment 2024 > Benign Liver Neoplasms

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eFigure 18–43. Hemangioma of the liver. Early post-contrast (M-7a) and late post-contrast (M-7b). Hemangiomas are the most common primary tumors of the liver, demonstrate progressive, centripetal contrast enhancement (arrows), and are uncommonly found in patients with cirrhosis. They are predominantly found in women and usually benign. (Used, with permission, from Nicholas Fidelman, MD). An axial early post-contrast image shows a tumor in a liver lobe. An axial late post-contrast image shows an opaque tumor in a liver lobe.

Current Medical Diagnosis & Treatment 2024 > Benign Liver Neoplasms

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 6–10. A: Ulcer on the nasal tip is the most obvious warning sign for this infiltrative BCC. B: Nodular BCC on the nasal ala of an 82-year-old woman. The nose is a very common location for a basal cell carcinoma. C: Large ulcerated nodular BCC on the mid-chest of a homeless man. The authors excised this in a free clinic located in a church basement. D: Large nodular basal cell carcinoma with an annular appearance on the face of a homeless woman. E: Superficial basal cell carcinoma on the back of a 45-year-old man who enjoys running in the California sun without his shirt. Note the diffuse scaling, thready border (slightly raised and pearly), and spotty hyperpigmentation on the edges. F: Large superficial BCC located on the back. Note the thready pearly border and small areas of pigmentation—both common features in superficial BCCs. G: Sclerosing BCC on the nose. Note the ivory white tumor that extends beyond the pearly pink segment. This took 4 stages of Mohs surgery to remove. H: Sclerosing and infiltrative basal cell carcinoma in a 45-year-old man diagnosed with a shave biopsy. The patient was sent for Mohs surgery because the BCC was aggressive and encroaching on the lower eyelid. (Reproduced with permission from Richard P. Usatine, MD, in Usatine RP, Smith MA, Mayeaux EJ Jr, Chumley HS. The Color Atlas and Synopsis of Family Medicine, 3rd ed. McGraw-Hill, 2019.) Eight photos show ulcers and basal cell carcinomas on the face.

Current Medical Diagnosis & Treatment 2024 > Basal Cell Carcinoma

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eFigure 28–21. A: Left infrarenal pheochromocytoma shown by CT scanning. B: Diagram identifies many of the visible structures. (Reproduced with permission from Greenspan FS, Strewler GJ. Basic & Clinical Endocrinology, 5th ed. McGraw-Hill Companies Inc.; 1997.) A scan shows the presence of a tumor between the bowel and vertebrae. An illustration shows the presence of a tumor between the bowel and vertebrae. Kidney, ureter, aorta, and vena cava are also identified.

Current Medical Diagnosis & Treatment 2024 > Pheochromocytoma & Paraganglioma

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eFigure 28–21. A: Left infrarenal pheochromocytoma shown by CT scanning. B: Diagram identifies many of the visible structures. (Reproduced with permission from Greenspan FS, Strewler GJ. Basic & Clinical Endocrinology, 5th ed. McGraw-Hill Companies Inc.; 1997.) A scan shows the presence of a tumor between the bowel and vertebrae. An illustration shows the presence of a tumor between the bowel and vertebrae. Kidney, ureter, aorta, and vena cava are also identified.

Current Medical Diagnosis & Treatment 2024 > Pheochromocytoma & Paraganglioma

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eFigure 41–1. An ulcerated mass with submucosal extension is visible in the mid-esophagus amidst columnar epithelium (Barrett esophagus). Biopsies confirmed adenocarcinoma. (Used, with permission, from Yao-Wen Cheng, MD.) An internal view of an esophagus shows a mass on the left side opening representing a mid esophagus adenocarcinoma tumor.

Current Medical Diagnosis & Treatment 2024 > Esophageal Cancer

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eFigure 41–6. A submucosal tumor with central ulceration. Fine-needle aspiration (FNA) confirmed GI stromal tumor (GIST). (Used, with permission, from Yao-Wen Cheng, MD.) A circular mass, a result of a gastrointestinal stromal tumor, G I S T. The center of the mass has a small indentation, caused by fine needle aspiration.

Current Medical Diagnosis & Treatment 2024 > Gastrointestinal Mesenchymal Tumors

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